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Allergies and Ulcerative Colitis

People with Ulcerative Colitis become hypersensitive to many substances because of the beating the body is taking and the inflammation issues that go with Ulcerative Colitis.

My son is allergic to many fruits and foods. Taking the supplement Quercetin does help to curb some of those. Every body is different so you will have to see if this helps you. I also have allergies and take this. It really helps me also. The brand below is what we take. It also contains Bromelain. Bromelain is a supplement that treats indigestion and reduces inflammation naturally.

My son also discovered that he can’t tolerate food dyes. At first you would think that food dyes are harmless because they are approved by the FDA but they are not. Red 40 and Yellow 5 and Yellow 6 food dyes cause stomach and intestinal upset and diarrhea in many people. Yellow 5 is actually banned in most European countries. Here is a good site: Check out the links on the side bar of the site for specific information on the dyes.

Not everything is known about allergic reactions in the body. What is known is that Mast Cells (responsible for symptoms of allergies) are involved and they are what release the powerful chemicals histamine and heparin.

An allergy is an attack of the Mast Cells with a histamine release, and raised IgA and IgG. IgA protects the body’s mucosal surfaces from infection including the mucosal lining in the colon. IgG can cause food allergies.

Many people with UC have high levels of histamine in their body including their colon. That is an indication that there is an allergic reaction. People with UC and sinusitis who are taking Claritin (an allergy medication) find that their UC symptoms return after they stop taking the medication. (28)

Here is a study that concludes: “Food elimination based on IgG antibodies may be effective in reducing IBS symptoms and is worthy of further biomedical research.”

Here is a study that states: “Patients with chronic ulcerative colitis (CUC) are known to have decreased spontaneous IgA secretion by colonic mononuclear cells. Considering the role of IgA, we propose that decreased mucosal IgA levels in CUC may predispose to the disease by a reduction of the immune-mediated exclusion mechanism and/or by an impairment of the down-regulation of the inflammatory response.”

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